C-Nap1 mutation affects centriole cohesion and is associated with a Seckel-like syndrome in cattle

نویسندگان

  • Sandrine Floriot
  • Christine Vesque
  • Sabrina Rodriguez
  • Florence Bourgain-Guglielmetti
  • Anthi Karaiskou
  • Mathieu Gautier
  • Amandine Duchesne
  • Sarah Barbey
  • Sébastien Fritz
  • Alexandre Vasilescu
  • Maud Bertaud
  • Mohammed Moudjou
  • Sophie Halliez
  • Valérie Cormier-Daire
  • Joyce E L Hokayem
  • Erich A Nigg
  • Luc Manciaux
  • Raphaël Guatteo
  • Nora Cesbron
  • Geraldine Toutirais
  • André Eggen
  • Sylvie Schneider-Maunoury
  • Didier Boichard
  • Joelle Sobczak-Thépot
  • Laurent Schibler
چکیده

Caprine-like Generalized Hypoplasia Syndrome (SHGC) is an autosomal-recessive disorder in Montbéliarde cattle. Affected animals present a wide range of clinical features that include the following: delayed development with low birth weight, hind limb muscular hypoplasia, caprine-like thin head and partial coat depigmentation. Here we show that SHGC is caused by a truncating mutation in the CEP250 gene that encodes the centrosomal protein C-Nap1. This mutation results in centrosome splitting, which neither affects centriole ultrastructure and duplication in dividing cells nor centriole function in cilium assembly and mitotic spindle organization. Loss of C-Nap1-mediated centriole cohesion leads to an altered cell migration phenotype. This discovery extends the range of loci that constitute the spectrum of autosomal primary recessive microcephaly (MCPH) and Seckel-like syndromes.

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C-Nap1, a Novel Centrosomal Coiled-Coil Protein and Candidate Substrate of the Cell Cycle–regulated Protein Kinase Nek2

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عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2015